The macrophage: the intersection between HIV infection and atherosclerosis

Suzanne Crowe; Clare Westhorpe; Nigora Mukhamedova; Anthony Jaworowski; Dimitri Sviridov; Michael Bukrinsky

doi:10.1189/jib.0809580

The macrophage: the intersection between HIV infection and atherosclerosis

Suzanne Crowe
, Clare Westhorpe
, Nigora Mukhamedova
, Anthony Jaworowski
, Dimitri Sviridov
, Michael Bukrinsky

Research output: Contribution to journal › Article › Research › peer-review

114 Citations (Scopus)

Abstract

HIV-infected individuals are at increased risj of coronary artery disease (CAD) with underlying mechanisms including chronic immune activation and inflammation secondary to HIV-induced microbial translocation and low-grade endotoxemia.

Original language	English
Pages (from-to)	589 - 598
Number of pages	10
Journal	Journal of Leukocyte Biology
Volume	87
DOIs	https://doi.org/10.1189/jib.0809580
Publication status	Published - 2010

UN SDGs

This output contributes to the following UN Sustainable Development Goals (SDGs)

SDG 3 Good Health and Well-being

Access to Document

10.1189/jib.0809580

Cite this

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title = "The macrophage: the intersection between HIV infection and atherosclerosis",

abstract = "HIV-infected individuals are at increased risj of coronary artery disease (CAD) with underlying mechanisms including chronic immune activation and inflammation secondary to HIV-induced microbial translocation and low-grade endotoxemia.",

author = "Suzanne Crowe and Clare Westhorpe and Nigora Mukhamedova and Anthony Jaworowski and Dimitri Sviridov and Michael Bukrinsky",

year = "2010",

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AU - Crowe, Suzanne

AU - Westhorpe, Clare

AU - Mukhamedova, Nigora

AU - Jaworowski, Anthony

AU - Sviridov, Dimitri

AU - Bukrinsky, Michael

PY - 2010

Y1 - 2010

N2 - HIV-infected individuals are at increased risj of coronary artery disease (CAD) with underlying mechanisms including chronic immune activation and inflammation secondary to HIV-induced microbial translocation and low-grade endotoxemia.

AB - HIV-infected individuals are at increased risj of coronary artery disease (CAD) with underlying mechanisms including chronic immune activation and inflammation secondary to HIV-induced microbial translocation and low-grade endotoxemia.

U2 - 10.1189/jib.0809580

DO - 10.1189/jib.0809580

M3 - Article

SN - 0741-5400

VL - 87

SP - 589

EP - 598

JO - Journal of Leukocyte Biology

JF - Journal of Leukocyte Biology

ER -