Sugar Fix Keeps RIPK3 at Bay

Cristina Giogha; Kate E. Lawlor

doi:10.1016/j.immuni.2019.02.018

Sugar Fix Keeps RIPK3 at Bay

Cristina Giogha, Kate E. Lawlor

Hudson Institute - Department of Molecular and Translational Science

Research output: Contribution to journal › Short Survey › Other › peer-review

4 Citations (Scopus)

Abstract

Immunometabolism is emerging as an important modulator of immune responses. In this issue of Immunity, Li et al. (2019) examine the link between lipopolysaccharide (LPS)-induced glucose metabolism and innate immune signaling and identify how β-N-acetylglucosamine (O-GlcNAc) modification of the RIPK3 RHIM domain limits inflammation and necroptosis.

Original language	English
Pages (from-to)	539-541
Number of pages	3
Journal	Immunity
Volume	50
Issue number	3
DOIs	https://doi.org/10.1016/j.immuni.2019.02.018
Publication status	Published - 19 Mar 2019

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10.1016/j.immuni.2019.02.018

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@article{4892b24d53974f21957baae4cfcd929c,

title = "Sugar Fix Keeps RIPK3 at Bay",

abstract = "Immunometabolism is emerging as an important modulator of immune responses. In this issue of Immunity, Li et al. (2019) examine the link between lipopolysaccharide (LPS)-induced glucose metabolism and innate immune signaling and identify how β-N-acetylglucosamine (O-GlcNAc) modification of the RIPK3 RHIM domain limits inflammation and necroptosis.",

author = "Cristina Giogha and Lawlor, {Kate E.}",

year = "2019",

month = mar,

day = "19",

doi = "10.1016/j.immuni.2019.02.018",

language = "English",

volume = "50",

pages = "539--541",

journal = "Immunity",

issn = "1074-7613",

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number = "3",

}

TY - JOUR

T1 - Sugar Fix Keeps RIPK3 at Bay

AU - Giogha, Cristina

AU - Lawlor, Kate E.

PY - 2019/3/19

Y1 - 2019/3/19

N2 - Immunometabolism is emerging as an important modulator of immune responses. In this issue of Immunity, Li et al. (2019) examine the link between lipopolysaccharide (LPS)-induced glucose metabolism and innate immune signaling and identify how β-N-acetylglucosamine (O-GlcNAc) modification of the RIPK3 RHIM domain limits inflammation and necroptosis.

AB - Immunometabolism is emerging as an important modulator of immune responses. In this issue of Immunity, Li et al. (2019) examine the link between lipopolysaccharide (LPS)-induced glucose metabolism and innate immune signaling and identify how β-N-acetylglucosamine (O-GlcNAc) modification of the RIPK3 RHIM domain limits inflammation and necroptosis.

UR - http://www.scopus.com/inward/record.url?scp=85062304142&partnerID=8YFLogxK

U2 - 10.1016/j.immuni.2019.02.018

DO - 10.1016/j.immuni.2019.02.018

M3 - Short Survey

AN - SCOPUS:85062304142

SN - 1074-7613

VL - 50

SP - 539

EP - 541

JO - Immunity

JF - Immunity

IS - 3

ER -

Sugar Fix Keeps RIPK3 at Bay

Abstract

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