No association between the Eczema genes COL29A1 and IL31 and inflammatory bowel disease

Marco Zucchelli; Leif Torkvist; Francesca Bresso; Jonas Halfvarson; Cilla Söderhäll; Young Ae Lee; Robert Löfberg; Juha Kere; Mauro D'Amato

doi:10.1002/ibd.20749

No association between the Eczema genes COL29A1 and IL31 and inflammatory bowel disease

Marco Zucchelli, Leif Torkvist, Francesca Bresso, Jonas Halfvarson, Cilla Söderhäll, Young Ae Lee, Robert Löfberg, Juha Kere, Mauro D'Amato

Research output: Contribution to journal › Letter › Other › peer-review

3 Citations (Scopus)

Abstract

Common genes and pathways have been discovered that affect individual predisposition to inflammatory diseases of the barrier organs such as asthma, eczema, psoriasis, and the inflammatory bowel diseases (IBDs), Crohn's disease (CD) and ulcerative colitis (UC).1 The underlying common pathogenetic mechanisms appear to involve a breakdown of the epithelial barrier (in the lungs, skin, or intestine), which leads to increased exposure to environmental stimuli (for instance, bacteria and allergens), and consequent dysregulation of the immune response in predisposed individuals.2

Original language	English
Pages (from-to)	961-962
Number of pages	2
Journal	Inflammatory Bowel Diseases
Volume	15
Issue number	7
DOIs	https://doi.org/10.1002/ibd.20749
Publication status	Published - 18 Sep 2009
Externally published	Yes

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title = "No association between the Eczema genes COL29A1 and IL31 and inflammatory bowel disease",

abstract = "Common genes and pathways have been discovered that affect individual predisposition to inflammatory diseases of the barrier organs such as asthma, eczema, psoriasis, and the inflammatory bowel diseases (IBDs), Crohn's disease (CD) and ulcerative colitis (UC).1 The underlying common pathogenetic mechanisms appear to involve a breakdown of the epithelial barrier (in the lungs, skin, or intestine), which leads to increased exposure to environmental stimuli (for instance, bacteria and allergens), and consequent dysregulation of the immune response in predisposed individuals.2",

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T1 - No association between the Eczema genes COL29A1 and IL31 and inflammatory bowel disease

AU - Zucchelli, Marco

AU - Torkvist, Leif

AU - Bresso, Francesca

AU - Halfvarson, Jonas

AU - Söderhäll, Cilla

AU - Lee, Young Ae

AU - Löfberg, Robert

AU - Kere, Juha

AU - D'Amato, Mauro

PY - 2009/9/18

Y1 - 2009/9/18

N2 - Common genes and pathways have been discovered that affect individual predisposition to inflammatory diseases of the barrier organs such as asthma, eczema, psoriasis, and the inflammatory bowel diseases (IBDs), Crohn's disease (CD) and ulcerative colitis (UC).1 The underlying common pathogenetic mechanisms appear to involve a breakdown of the epithelial barrier (in the lungs, skin, or intestine), which leads to increased exposure to environmental stimuli (for instance, bacteria and allergens), and consequent dysregulation of the immune response in predisposed individuals.2

AB - Common genes and pathways have been discovered that affect individual predisposition to inflammatory diseases of the barrier organs such as asthma, eczema, psoriasis, and the inflammatory bowel diseases (IBDs), Crohn's disease (CD) and ulcerative colitis (UC).1 The underlying common pathogenetic mechanisms appear to involve a breakdown of the epithelial barrier (in the lungs, skin, or intestine), which leads to increased exposure to environmental stimuli (for instance, bacteria and allergens), and consequent dysregulation of the immune response in predisposed individuals.2

UR - http://www.scopus.com/inward/record.url?scp=69449089354&partnerID=8YFLogxK

U2 - 10.1002/ibd.20749

DO - 10.1002/ibd.20749

M3 - Letter

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AN - SCOPUS:69449089354

VL - 15

SP - 961

EP - 962

JO - Inflammatory Bowel Diseases

JF - Inflammatory Bowel Diseases

SN - 1078-0998

IS - 7

ER -

No association between the Eczema genes COL29A1 and IL31 and inflammatory bowel disease

Abstract

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