Abstract
In this issue of Blood, Madhusudhan et al elegantly demonstrate a novel role of the coagulation protease activated protein C (aPC) in maintaining endoplasmic reticulum (ER) homeostasis, and thus cellular metabolism, in the context of diabetic kidney disease (DKD).1
Original language | English |
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Pages (from-to) | 1390-1391 |
Number of pages | 2 |
Journal | Blood |
Volume | 130 |
Issue number | 12 |
DOIs |
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Publication status | Published - 21 Sept 2017 |