An event of shear importance

Robert Andrews, Michael Berndt

Research output: Contribution to journalLetterOther

2 Citations (Scopus)

Abstract

In this issue of Blood, Yong and colleagues report that circulating blood platelets and monocytes in stable coronary artery disease patients can become activated by shear stress during passage through a stenosed sclerotic lesion, despite dual anti-platelet therapy, and the absence of either plaque rupture or collagen exposure.
Original languageEnglish
Pages (from-to)5 - 7
Number of pages3
JournalBlood
Volume117
Issue number1
DOIs
Publication statusPublished - 2011

Cite this

Andrews, Robert ; Berndt, Michael. / An event of shear importance. In: Blood. 2011 ; Vol. 117, No. 1. pp. 5 - 7.
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abstract = "In this issue of Blood, Yong and colleagues report that circulating blood platelets and monocytes in stable coronary artery disease patients can become activated by shear stress during passage through a stenosed sclerotic lesion, despite dual anti-platelet therapy, and the absence of either plaque rupture or collagen exposure.",
author = "Robert Andrews and Michael Berndt",
year = "2011",
doi = "10.1182/blood-2010-10-312116",
language = "English",
volume = "117",
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journal = "Blood",
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An event of shear importance. / Andrews, Robert; Berndt, Michael.

In: Blood, Vol. 117, No. 1, 2011, p. 5 - 7.

Research output: Contribution to journalLetterOther

TY - JOUR

T1 - An event of shear importance

AU - Andrews, Robert

AU - Berndt, Michael

PY - 2011

Y1 - 2011

N2 - In this issue of Blood, Yong and colleagues report that circulating blood platelets and monocytes in stable coronary artery disease patients can become activated by shear stress during passage through a stenosed sclerotic lesion, despite dual anti-platelet therapy, and the absence of either plaque rupture or collagen exposure.

AB - In this issue of Blood, Yong and colleagues report that circulating blood platelets and monocytes in stable coronary artery disease patients can become activated by shear stress during passage through a stenosed sclerotic lesion, despite dual anti-platelet therapy, and the absence of either plaque rupture or collagen exposure.

U2 - 10.1182/blood-2010-10-312116

DO - 10.1182/blood-2010-10-312116

M3 - Letter

VL - 117

SP - 5

EP - 7

JO - Blood

JF - Blood

SN - 0006-4971

IS - 1

ER -