Alternatively activated macrophage-derived RELM-α is a negative regulator of type 2 inflammation in the lung (Journal of Experimental Medicine (2009) 206, 4, (937-952))

Meera G. Nair; Yurong Du; Jacqueline G. Perrigoue; Colby Zaph; Justin J. Taylor; Michael Goldschmidt; Gary P. Swain; George D. Yancopoulos; David M. Valenzuela; Andrew Murphy; Margaret Karow; Sean Stevens; Edward J. Pearce; David Artis

doi:10.1084/jem.2008204841009c

Alternatively activated macrophage-derived RELM-α is a negative regulator of type 2 inflammation in the lung (Journal of Experimental Medicine (2009) 206, 4, (937-952))

Meera G. Nair, Yurong Du, Jacqueline G. Perrigoue, Colby Zaph, Justin J. Taylor, Michael Goldschmidt, Gary P. Swain, George D. Yancopoulos, David M. Valenzuela, Andrew Murphy, Margaret Karow, Sean Stevens, Edward J. Pearce, David Artis

Biochemistry & Molecular Biology

Research output: Contribution to journal › Comment / Debate › Other › peer-review

10 Citations (Scopus)

Original language	English
Pages (from-to)	1201
Number of pages	1
Journal	Journal of Experimental Medicine
Volume	206
Issue number	5
DOIs	https://doi.org/10.1084/jem.2008204841009c
Publication status	Published - 11 May 2009

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10.1084/jem.2008204841009c

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Nair, M. G., Du, Y., Perrigoue, J. G., Zaph, C., Taylor, J. J., Goldschmidt, M., Swain, G. P., Yancopoulos, G. D., Valenzuela, D. M., Murphy, A., Karow, M., Stevens, S., Pearce, E. J., & Artis, D. (2009). Alternatively activated macrophage-derived RELM-α is a negative regulator of type 2 inflammation in the lung (Journal of Experimental Medicine (2009) 206, 4, (937-952)). Journal of Experimental Medicine, 206(5), 1201. https://doi.org/10.1084/jem.2008204841009c

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title = "Alternatively activated macrophage-derived RELM-α is a negative regulator of type 2 inflammation in the lung (Journal of Experimental Medicine (2009) 206, 4, (937-952))",

author = "Nair, \{Meera G.\} and Yurong Du and Perrigoue, \{Jacqueline G.\} and Colby Zaph and Taylor, \{Justin J.\} and Michael Goldschmidt and Swain, \{Gary P.\} and Yancopoulos, \{George D.\} and Valenzuela, \{David M.\} and Andrew Murphy and Margaret Karow and Sean Stevens and Pearce, \{Edward J.\} and David Artis",

year = "2009",

month = may,

day = "11",

doi = "10.1084/jem.2008204841009c",

language = "English",

volume = "206",

pages = "1201",

journal = "Journal of Experimental Medicine",

issn = "0022-1007",

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Nair, MG, Du, Y, Perrigoue, JG, Zaph, C, Taylor, JJ, Goldschmidt, M, Swain, GP, Yancopoulos, GD, Valenzuela, DM, Murphy, A, Karow, M, Stevens, S, Pearce, EJ & Artis, D 2009, 'Alternatively activated macrophage-derived RELM-α is a negative regulator of type 2 inflammation in the lung (Journal of Experimental Medicine (2009) 206, 4, (937-952))', Journal of Experimental Medicine, vol. 206, no. 5, pp. 1201. https://doi.org/10.1084/jem.2008204841009c

Alternatively activated macrophage-derived RELM-α is a negative regulator of type 2 inflammation in the lung (Journal of Experimental Medicine (2009) 206, 4, (937-952)). / Nair, Meera G.; Du, Yurong; Perrigoue, Jacqueline G. et al.
In: Journal of Experimental Medicine, Vol. 206, No. 5, 11.05.2009, p. 1201.

Research output: Contribution to journal › Comment / Debate › Other › peer-review

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T1 - Alternatively activated macrophage-derived RELM-α is a negative regulator of type 2 inflammation in the lung (Journal of Experimental Medicine (2009) 206, 4, (937-952))

AU - Nair, Meera G.

AU - Du, Yurong

AU - Perrigoue, Jacqueline G.

AU - Zaph, Colby

AU - Taylor, Justin J.

AU - Goldschmidt, Michael

AU - Swain, Gary P.

AU - Yancopoulos, George D.

AU - Valenzuela, David M.

AU - Murphy, Andrew

AU - Karow, Margaret

AU - Stevens, Sean

AU - Pearce, Edward J.

AU - Artis, David

PY - 2009/5/11

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U2 - 10.1084/jem.2008204841009c

DO - 10.1084/jem.2008204841009c

M3 - Comment / Debate

AN - SCOPUS:66049102548

SN - 0022-1007

VL - 206

SP - 1201

JO - Journal of Experimental Medicine

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IS - 5

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Alternatively activated macrophage-derived RELM-α is a negative regulator of type 2 inflammation in the lung (Journal of Experimental Medicine (2009) 206, 4, (937-952))

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