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Projects
- 1 Active
Research Output
Correction: NK cell-derived GM-CSF potentiates inflammatory arthritis and is negatively regulated by CIS
Louis, C., Souza-Fonseca-Guimaraes, F., Yang, Y., D'Silva, D., Kratina, T., Dagley, L., Hediyeh-Zadeh, S., Rautela, J., Masters, S. L., Davis, M. J., Babon, J. J., Ciric, B., Vivier, E., Alexander, W. S., Huntington, N. D. & Wicks, I. P., 4 May 2020, In : Journal of Experimental Medicine. 217, 5, 1 p.Research output: Contribution to journal › Article › Other › peer-review
Open Access
File
miR17~92 restrains pro-apoptotic BIM to ensure survival of haematopoietic stem and progenitor cells
Brinkmann, K., Ng, A. P., de Graaf, C. A., Di Rago, L., Hyland, C. D., Morelli, E., Rautela, J., Huntington, N. D., Strasser, A., Alexander, W. S. & Herold, M. J., May 2020, In : Cell Death and Differentiation. 27, 5, p. 1475-1488 14 p.Research output: Contribution to journal › Article › Research › peer-review
Open Access
File
NK cell–derived GM-CSF potentiates inflammatory arthritis and is negatively regulated by CIS
Louis, C., Souza-Fonseca-Guimaraes, F., Yang, Y., D'Silva, D., Kratina, T., Dagley, L., Hediyeh-Zadeh, S., Rautela, J., Masters, S. L., Davis, M. J., Babon, J. J., Ciric, B., Vivier, E., Alexander, W. S., Huntington, N. D. & Wicks, I. P., 4 May 2020, In : Journal of Experimental Medicine. 217, 5, 19 p., e20191421.Research output: Contribution to journal › Article › Research › peer-review
Open Access
File
1
Citation
(Scopus)
NK Cell Priming From Endogenous Homeostatic Signals Is Modulated by CIS
Delconte, R. B., Guittard, G., Goh, W., Hediyeh-Zadeh, S., Hennessy, R. J., Rautela, J., Davis, M. J., Souza-Fonseca-Guimaraes, F., Nunès, J. A. & Huntington, N. D., 31 Jan 2020, In : Frontiers in Immunology. 11, 16 p., 75.Research output: Contribution to journal › Article › Research › peer-review
Open Access
File
1
Citation
(Scopus)
The cancer–natural killer cell immunity cycle
Huntington, N. D., Cursons, J. & Rautela, J., 24 Jun 2020, (Accepted/In press) In : Nature Reviews Cancer. 18 p.Research output: Contribution to journal › Review Article › Research › peer-review