Projects per year
Personal profile
Research interests
Cell signalling in renal inflammation and fibrosis
Biography
Dr Ma graduated from the Joint 8-year MD Program of Nankai University and Tianjin Medical University, China in 1989. He then worked as a surgeon in Tianjin Institute of Urology, the Second Affiliated Hospital of Tianjin Medical University for 9 years from 1989 to 1998. After moving to New Zealand and received his PhD from the University of Otago in 2004, Dr Ma commenced his research in kidney disease as a research fellow in the Department of Nephrology at Monash Medical Centre, Australia.
Expertise related to UN Sustainable Development Goals
In 2015, UN member states agreed to 17 global Sustainable Development Goals (SDGs) to end poverty, protect the planet and ensure prosperity for all. This person’s work contributes towards the following SDG(s):
Education/Academic qualification
Medical Science, PhD, Regulation of Prolactin in hypothalamus neurons, University of Otago
2000 → 2004
Award Date: 16 Jun 2004
Medicine, MD, Joint 8-Year MD Program, Nankai University and Tianjin Medical University, Tianjin Medical University
1981 → 1989
Award Date: 18 Aug 1989
Research area keywords
- kidney disease
- Fibrosis
- Inflammation
- Kinase Inhibitors
- transplantation
Collaborations and top research areas from the last five years
Recent external collaboration on country/territory level. Dive into details by clicking on the dots or
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Improving short- and long-term outcomes in cardiac bypass surgery by preventing acute kidney injury.
Nikolic-Paterson, D., Ma, F., Smith, J., Grynberg, K., Mulley, W., Robertson, T. & Combes, A.
1/06/22 → 31/05/24
Project: Research
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Therapeutic targeting of cellular senescence to prevent progression of chronic kidney disease
Tesch, G., Nikolic-Paterson, D. & Ma, F.
3/05/21 → 31/03/22
Project: Research
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Use of an early urine biomarker to identify individuals who can best benefit from ASK1 inhibitor therapy during diabetes.
Tesch, G., Nikolic-Paterson, D. & Ma, F.
3/05/21 → 3/05/22
Project: Research
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Overcoming Vulnerability and Improving Outcomes of Acute Kidney Injury in Diabetic Patients
18/03/21 → 17/03/22
Project: Research
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Preventing and Limiting Acute Kidney Injury in High Risk Diabetic Patients
Nikolic-Paterson, D., Ma, F. & Tesch, G.
1/01/19 → 31/12/19
Project: Other
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Corrigendum: c-Jun amino terminal kinase signaling promotes aristolochic acid-induced acute kidney injury, (Front. Physiol, (2021), 12, (599114), 10.3389/fphys.2021.599114)
Yang, F., Ozols, E., Ma, F. Y., Leong, K. G., Tesch, G. H., Jiang, X. & Nikolic-Paterson, D. J., 4 Jan 2023, In: Frontiers in Physiology. 13, 1 p., 1123475.Research output: Contribution to journal › Comment / Debate › Other › peer-review
Open Access -
Mice with Established Diabetes Show Increased Susceptibility to Renal Ischemia/Reperfusion Injury: Protection by Blockade of Jnk or Syk Signaling Pathways
Grynberg, K., Tian, L., Tesch, G., Ozols, E., Mulley, W. R., Nikolic-Paterson, D. J. & Ma, F. Y., Mar 2022, In: American Journal of Pathology. 192, 3, p. 441-453 13 p.Research output: Contribution to journal › Article › Research › peer-review
2 Citations (Scopus) -
c-Jun Amino Terminal Kinase Signaling Promotes Aristolochic Acid-Induced Acute Kidney Injury
Yang, F., Ozols, E., Ma, F. Y., Leong, K. G., Tesch, G. H., Jiang, X. & Nikolic-Paterson, D. J., 12 Feb 2021, In: Frontiers in Physiology. 12, 12 p., 599114.Research output: Contribution to journal › Article › Research › peer-review
Open Access5 Citations (Scopus) -
Cyclophilin D promotes acute, but not chronic, kidney injury in a mouse model of aristolochic acid toxicity
Leong, K. G., Ozols, E., Kanellis, J., Ma, F. Y. & Nikolic-Paterson, D. J., Oct 2021, In: Toxins. 13, 10, 13 p., 700.Research output: Contribution to journal › Article › Research › peer-review
Open Access2 Citations (Scopus) -
Cyclophilin inhibition protects against experimental acute kidney injury and renal interstitial fibrosis
Leong, K. G., Ozols, E., Kanellis, J., Badal, S. S., Liles, J. T., Nikolic-Paterson, D. J. & Ma, F. Y., Jan 2021, In: International Journal of Molecular Sciences. 22, 1, 17 p., 271.Research output: Contribution to journal › Article › Research › peer-review
Open Access11 Citations (Scopus)